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PA H9 | Spleen Disorders — Practice Quiz

Practice 10 questions · Untimed · Unlimited attempts

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Q1 PA19.4 1 pt

Which of the following is NOT a recognised function of the normal spleen?

A Filtration and destruction of senescent red blood cells
B Production of opsonins (properdin and tuftsin)
C Synthesis of clotting factor VIII
D Reservoir function for platelets and granulocytes

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Q2 PA19.4 1 pt

A 35-year-old man presents with gradual-onset massive splenomegaly, anaemia, and hepatomegaly. Bone marrow biopsy shows a 'dry tap', and trephine reveals hypercellular marrow with prominent reticulin fibrosis and immature myeloid precursors. The most likely diagnosis is:

A Chronic myeloid leukaemia (CML)
B Primary myelofibrosis
C Kala-azar (visceral leishmaniasis)
D β-thalassaemia major

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Q3 PA19.4 1 pt

Which mechanism best explains the splenomegaly seen in portal hypertension due to hepatic cirrhosis?

A Infiltration of the splenic parenchyma by macrophages laden with storage material
B Passive congestion due to impaired venous drainage from the spleen
C Reactive lymphoid hyperplasia in response to portal bacteraemia
D Extramedullary haematopoiesis compensating for bone marrow failure

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Q4 PA19.4 1 pt

Hypersplenism is defined as:

A Splenomegaly with peripheral cytopenias that improve after splenectomy
B Absolute spleen weight exceeding 500 g with any cytopenia
C Splenomegaly causing portal hypertension via splenic vein compression
D Excessive splenic production of immunoglobulins causing haemolysis

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Q5 PA19.4 1 pt

A peripheral blood smear from a 28-year-old who underwent splenectomy three years ago for hereditary spherocytosis shows red cells containing small, dense nuclear remnants. These inclusions are:

A Heinz bodies (denatured haemoglobin)
B Howell-Jolly bodies (nuclear remnants)
C Pappenheimer bodies (iron granules)
D Cabot rings (remnant spindle fibre)

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Q6 PA19.4 1 pt

Following splenectomy, patients are at highest risk for overwhelming post-splenectomy infection (OPSI) from which group of organisms?

A Intracellular organisms such as Mycobacterium tuberculosis and Listeria
B Encapsulated bacteria such as Streptococcus pneumoniae, Haemophilus influenzae, and Neisseria meningitidis
C Gram-negative enteric bacilli such as Escherichia coli and Klebsiella
D Anaerobes such as Bacteroides fragilis and Clostridium species

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Q7 PA19.4 1 pt

A 19-year-old woman from Bihar presents with 3-month history of fever, weight loss, and massive splenomegaly. Serology is positive for anti-rK39 antibody. Bone marrow aspirate shows macrophages laden with oval, non-flagellated organisms (2–4 µm). The causative vector is:

A Anopheles mosquito
B Female Phlebotomus sandfly
C Ixodes tick
D Culex mosquito

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Q8 PA19.4 1 pt

Which of the following pairings of disease and mechanism of splenomegaly is CORRECT?

A Infective endocarditis — infiltrative (tumour cells)
B Systemic lupus erythematosus (SLE) — immune (lymphoid hyperplasia and antibody production)
C Gaucher disease — congestive (sinusoidal congestion)
D Beta-thalassaemia major — infective (parasitic infiltration)

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Q9 PA19.4 1 pt

A 12-year-old boy from Odisha presents in June with high-grade fever, rigors, and massive splenomegaly. His blood smear shows RBCs with multiple ring-form trophozoites and banana-shaped gametocytes. Which complication is MOST life-threatening in this form of malaria?

A Spontaneous splenic rupture
B Cerebral malaria from sequestration of parasitised RBCs in cerebral microvessels
C Blackwater fever from immune-complex glomerulonephritis
D Burkitt lymphoma from EBV reactivation

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Q10 PA19.4 1 pt

A 16-year-old boy with known β-thalassaemia major on regular blood transfusions develops progressive splenomegaly with increasing transfusion requirements (from 2-unit every 4 weeks to every 2 weeks). Serum ferritin is 4,200 ng/mL. Which is the MOST likely cause of his increasing transfusion requirement?

A Iron-overload cardiomyopathy reducing cardiac output
B Hypersplenism causing sequestration and destruction of transfused red cells
C Alloimmunisation developing antibodies against transfused red cell antigens
D Ineffective erythropoiesis worsening due to iron toxicity to erythroid progenitors

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